Background and details
Smoking is one of the most important risk factors for the development and progression of periodontal disease. Smoking patients are up to 3 times more likely to develop periodontitis than non-smokers: this leads to greater bone loss and increased rectitration ("retactions") of their gums.
Smokers also responded worse to periodontal treatment versus non-smokers. Even the rate of periodontitis progression is higher in smokers but reduced if the patient stops smoking and recent former smokers have responded to periodontitis treatment similar to patients who have never smoked. Smoking should visit the periodicist more often than non-smokers.
The increased risk of periodontitis among smokers is due, among other reasons, to more pathogenic bacteria in their mouth. Smoking is also with alcohol, the leading cause of oral cancer. To the bad effect of toxic tobacco products, the effect of smoking-induced heat binds. The smoking of cigarettes includes about 4,000 constituents that are pharmacologically toxic, mutagenic and carcinogenic. Smokers have higher levels of pathogenic bacteria in the periodite and have a decline in gum defences as a result of the bacterial attack.
It is estimated that the number of failures after treatment with dental implants in smoking patients may be twice as high as in non-smokers, so it's highly advisable to leave this habit if implant placement is being planted. If this cannot be achieved, at least smoking should be stopped from two weeks before intervention up to eight weeks after intervention.
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